Search results for "Phosphodiesterase inhibitor"

showing 10 items of 78 documents

Selective Inhibition of Phosphodiesterases 4A, B, C and D Isoforms in Chronic Respiratory Diseases: Current and Future Evidences

2016

Chronic respiratory diseases affect millions of people every day. According to the World Health Organization estimates, ~235 million people suffer from asthma, ~64 million suffer from chronic obstructive pulmonary disease (COPD), and millions more suffer from allergic rhinitis around the world. In recent last years, the first phosphodiesterase 4 (PDE4) inhibitor, roflumilast, was approved as a treatment to reduce the risk of exacerbations in stable and severe COPD associated with chronic bronchitis and a history of exacerbations. PDE4 exists as four subtypes (A, B, C, and D) each with a capacity to degrade cAMP, a second messenger involved in inflammatory responses. PDE4 inhibitors inhibit …

0301 basic medicineChronic bronchitisPhosphodiesterase InhibitorsPharmacologyPulmonary Disease Chronic Obstructive03 medical and health sciencesPDE4BDrug DiscoverymedicineAnimalsHumansRespiratory systemRoflumilastAsthmaPharmacologyCOPDPhosphoric Diester Hydrolasesbusiness.industryPhosphodiesterasemedicine.diseaseIsoenzymesDiarrhea030104 developmental biologymedicine.symptombusinessmedicine.drugCurrent Pharmaceutical Design
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The β3 -adrenoceptor agonist mirabegron increases human atrial force through β1 -adrenoceptors: an indirect mechanism?

2017

Background and Purpose Mirabegron has been classified as a β3-adrenoceptor agonist approved for overactive bladder syndrome. We investigated possible cardiac effects of mirabegron in the absence or presence of β-adrenoceptor subtype antagonists. In view of its phenylethanolamine structure, we investigated whether mirabegron has indirect sympathomimetic activity by using neuronal uptake blockers. Experimental Approach Right atrial trabeculae, from non-failing hearts, were paced and contractile force measured at 37°C. Single concentrations of mirabegron were added in the absence or presence of the phosphodiesterase inhibitor 3-isobutyl-1-methylxanthine (IBMX), β3 (L-748,337), β1 (CGP 20712A),…

0301 basic medicinePharmacologyAgonistmedicine.medical_specialtyIBMXContraction (grammar)PhenoxybenzamineChemistrymedicine.drug_classAdrenergicPharmacology03 medical and health scienceschemistry.chemical_compound030104 developmental biologyEndocrinologyInternal medicineDesipraminemedicinePhosphodiesterase inhibitorMirabegronmedicine.drugBritish Journal of Pharmacology
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Pentoxifylline Inhibits Vγ9/Vδ2 T Lymphocyte Activation of Patients with Active Behçet's Disease in Vitro

2007

The aim of this study is to evaluate the in vitro effect of pentoxifylline (PTX) on T Vgamma9/Vdelta2 lymphocyte function in Behçets disease (BD). We investigated the effect of PTX on Vgamma9/Vdelta2 T cell expansion and expression of TNFRII receptor and perforin content before and after PTX addition by means of FACS analysis lymphocyte cultures from patients with active and inactive BD and healthy subjects. The addition of PTX at a concentration of 1 mg/ml determined a significant inhibition of cell expansion, a down regulation of TNF receptor expression and inhibited the PMA-induced degranulation of perforin. Taken together these data indicate that PTX is capable of interfering with Vgamm…

AdultMaleCytoplasmPhosphodiesterase InhibitorsT-LymphocytesT cellLymphocyteImmunologyIn Vitro TechniquesPharmacologyLymphocyte ActivationPentoxifylline03 medical and health sciences0302 clinical medicineIn vivomedicineHumansReceptors Tumor Necrosis Factor Type IIImmunology and AllergyPentoxifyllineReceptorPharmacologybiologyPerforinChemistryBehcet SyndromeDegranulationReceptors Antigen T-Cell gamma-deltaFlow Cytometrymedicine.anatomical_structurePerforinCell culture030220 oncology & carcinogenesisLeukocytes Mononuclearbiology.proteinFemale030215 immunologymedicine.drugInternational Journal of Immunopathology and Pharmacology
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Relaxation and cyclic GMP levels in response to sildenafil in human pulmonary arteries from donors.

2005

We measured cyclic GMP formation and relaxation response to sildenafil given either alone or in combination with sodium nitroprusside (SNP) in pulmonary arteries obtained from 13 multi-organ donors. Sildenafil (10(-9)-10(-4) M) caused concentration-dependent relaxations and amplified the relaxation induced by SNP. Relaxation was unaffected by endothelium removal or by pre-treatment with the inhibitor of nitric oxide synthase L-NMMA (10(-4) M). SNP (10(-7) M) caused elevation of cyclic GMP levels that was potentiated by sildenafil (10(-6) M). Thus, the enhancement of SNP-induced relaxation by sildenafil is mainly due to an increase in cyclic GMP accumulation.

AdultMaleNitroprussideEndotheliumSildenafilPhosphodiesterase InhibitorsVasodilator AgentsVasodilationPharmacologyIn Vitro TechniquesPulmonary ArteryPiperazinesSildenafil CitrateNitric oxidechemistry.chemical_compound3'5'-Cyclic-GMP PhosphodiesterasesmedicineHumansNitric Oxide DonorsSulfonesCyclic GMPPharmacologybiologyChemistryDrug SynergismMiddle Agedrespiratory tract diseasesNitric oxide synthaseVasodilationmedicine.anatomical_structureBiochemistryEnzyme inhibitorPurinesCirculatory systemcardiovascular systembiology.proteinFemaleSodium nitroprussidemedicine.drugEuropean journal of pharmacology
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Characterization of collagenase 3 (matrix metalloproteinase 13) messenger RNA expression in the synovial membrane and synovial fibroblasts of patient…

1999

Objective To study the localization and cell type–specific expression of collagenase 3 messenger RNA (mRNA) in the synovial membrane, its regulation in primary synovial fibroblasts, and the correlation with systemic markers of inflammation and radiographic damage in rheumatoid arthritis (RA). Methods The expression of collagenase 3 mRNA was characterized by Northern blot analysis, reverse transcriptase–polymerase chain reaction, and in situ hybridization. Immunohistochemical detection of cell type–specific antigens was used in combination with in situ hybridization of collagenase 3 mRNA to characterize the cellular origin of collagenase 3 mRNA expression. Results Collagenase 3 mRNA was dete…

AdultMalePathologymedicine.medical_specialtyPhosphodiesterase InhibitorsImmunologyIn situ hybridizationBiologyArthritis RheumatoidRheumatology1-Methyl-3-isobutylxanthineMatrix Metalloproteinase 13Cyclic AMPmedicineHumansImmunology and AllergyPharmacology (medical)CollagenasesRNA MessengerNorthern blotFibroblastCells CulturedIn Situ HybridizationAgedAged 80 and overMessenger RNAColforsinSynovial MembraneFibroblastsMiddle AgedMolecular biologyEnzyme ActivationRadiographymedicine.anatomical_structureBucladesineGene Expression RegulationCell cultureCollagenaseInterstitial collagenaseFemaleSynovial membraneAdenylyl Cyclasesmedicine.drugArthritis & Rheumatism
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Methazolamide Plus Aminophylline Abrogates Hypoxia-Mediated Endurance Exercise Impairment.

2015

In hypoxia, endurance exercise performance is diminished; pharmacotherapy may abrogate this performance deficit. Based on positive outcomes in preclinical trials, we hypothesized that oral administration of methazolamide, a carbonic anhydrase inhibitor, aminophylline, a nonselective adenosine receptor antagonist and phosphodiesterase inhibitor, and/or methazolamide combined with aminophylline would attenuate hypoxia-mediated decrements in endurance exercise performance in humans. Fifteen healthy males (26 ± 5 years, body-mass index: 24.9 ± 1.6 kg/m(2); mean ± SD) were randomly assigned to one of four treatments: placebo (n = 9), methazolamide (250 mg; n = 10), aminophylline (400 mg; n = 9),…

AdultMalePhysiologymedicine.drug_classMethazolamideAdenosine receptor antagonistPlaceboYoung AdultEndurance trainingmedicineHumansCarbonic anhydrase inhibitorPhosphodiesterase inhibitorMethazolamideHypoxiaExercisebusiness.industryAltitudePublic Health Environmental and Occupational HealthGeneral MedicineHypoxia (medical)AminophyllineHealthy VolunteersAnesthesiaExercise TestPhysical EnduranceAminophyllineDrug Therapy Combinationmedicine.symptombusinessmedicine.drugHigh altitude medicinebiology
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Effects of sildenafil on human penile blood vessels.

2000

Abstract Objectives. To investigate the effects of sildenafil on human penile blood vessels and evaluate the interaction of sildenafil with neurogenic-mediated responses. Sildenafil is currently used in the treatment of erectile dysfunction. Methods. Penile dorsal arteries and deep dorsal veins were obtained from 14 multiorgan donors. Vascular rings were suspended in organ bath chambers, and the isometric tension was recorded. We then studied the effects of sildenafil on precontracted vessels and the neurogenic (noradrenergic and nitrergic) responses. Results. Sildenafil (10 −9 to 3 × 10 −6 M) caused concentration-dependent relaxation and amplified the relaxation induced by sodium nitroprus…

AdultMalemedicine.medical_specialtyAdolescentSildenafilPhosphodiesterase InhibitorsUrologyMuscle RelaxationPiperazinesSildenafil CitrateNitric oxideVeinschemistry.chemical_compoundInternal medicineMedicineHumansSulfonesGuanethidinebusiness.industrySmooth muscle contractionArteriesMiddle AgedPDE5 drug designrespiratory tract diseasesVasodilationEndocrinologymedicine.anatomical_structurechemistryPurinesVasoconstrictioncardiovascular systemSodium nitroprussidebusinessZaprinastBlood vesselmedicine.drugMuscle ContractionPenisUrology
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Investigation into the role of phosphodiesterase IV in bronchorelaxation, including studies with human bronchus.

1993

1. We have investigated the role of cyclic nucleotide phosphodiesterase IV (PDE IV) in the relaxation of human bronchus and guinea-pig trachea in vitro and in guinea-pigs in vivo. 2. Functional studies showed that the selective PDE IV inhibitors, rolipram and denbufylline, relaxed human and guinea-pig preparations in vitro. 3. Two clinically used xanthine non-selective PDE inhibitors, theophylline and pentoxifylline, were also effective in these preparations, but were much less potent than the selective agents used. 4. The rank order of potency for the four PDE inhibitors in both species was similar. 5. Biochemical studies indicated that PDE IV was the major PDE isoform present in the human…

AdultMalemedicine.medical_specialtyPhosphodiesterase InhibitorsGuinea PigsBiologyIn Vitro TechniquesPentoxifyllinechemistry.chemical_compoundTheophyllineIn vivoInternal medicinemedicineAnimalsHumansTheophyllineheterocyclic compoundsPentoxifyllineRolipramAgedPharmacologyCyclic nucleotide phosphodiesterasePhosphoric Diester HydrolasesIsoproterenolMiddle AgedXanthinemusculoskeletal systemAsthmaPyrrolidinonesBronchodilator AgentsCyclic Nucleotide Phosphodiesterases Type 4IsoenzymesBronchodilatationenzymes and coenzymes (carbohydrates)Disease Models AnimalEndocrinologychemistryEnzyme inhibitor3'5'-Cyclic-AMP PhosphodiesterasesXanthinesbiology.proteinFemalesense organsRoliprammedicine.drugcirculatory and respiratory physiologyResearch Article
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Relaxation induced by milrinone and rolipram in human penile arteries and veins

2002

Abstract We studied the relaxant effects of milrinone, an inhibitor of phosphodiesterase 3, and rolipram, an inhibitor of phosphodiesterase 4, on contracted human penile dorsal artery and deep dorsal vein. Vascular rings from 12 multi-organ donors were suspended in organ baths for isometric recording of tension. Both milrinone and rolipram inhibited (100%) the contraction induced by noradrenaline and shifted the relaxation–response curves to the cAMP forming agents prostaglandin E1 and forskolin to the left. The findings indicate that the cAMP pathway appears to be a main determinant of relaxation in human penile vessels.

AdultMalemedicine.medical_specialtyPhosphodiesterase InhibitorsPhosphodiesterase 3Penile arteryBiologyMuscle Smooth Vascularchemistry.chemical_compoundInternal medicinemedicineHumansDrug InteractionsChildProstaglandin E1RolipramPharmacologyForskolinDose-Response Relationship DrugColforsinMiddle AgedVasodilationEndocrinologymedicine.anatomical_structurechemistryCirculatory systemMilrinoneRolipramMilrinonePenisBlood vesselmedicine.drugEuropean Journal of Pharmacology
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Relaxation of the isolated human internal anal sphincter by sildenafil.

2007

Abstract Background Hypertonicity of the internal anal sphincter (IAS) appears to be involved in the pathogenesis of anal fissure. The relaxant effects of sildenafil, a selective phosphodiesterase 5 (PDE5) inhibitor, on isolated human IAS were investigated. Methods The efficacy (maximal effect, Emax) and potency (−log IC50, where IC50 is half-maximal inhibitory concentration) of the PDE5 inhibitors, sildenafil and zaprinast, and of nitric oxide donors, sodium nitroprusside and glyceryl trinitrate, as relaxants of histamine (0·1 mmol/l)-induced tone were examined in IAS strips under isometric contraction. The presence of PDE5 isoenzymes and changes in intracellular calcium and cyclic nucleot…

AdultMalemedicine.medical_specialtySildenafilPhosphodiesterase InhibitorsMuscle RelaxationAnal CanalIn Vitro TechniquesPiperazinesSildenafil CitrateInternal anal sphincterchemistry.chemical_compoundCyclic nucleotideInternal medicinemedicineHumansSulfonesCyclic GMPAgedAged 80 and overAnal fissureDose-Response Relationship Drugbusiness.industryMuscle SmoothMiddle Agedmedicine.diseaseMuscle relaxationEndocrinologychemistryPurinescGMP-specific phosphodiesterase type 5SurgeryFemaleSodium nitroprussideZaprinastbusinessmedicine.drugThe British journal of surgery
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